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- W2056788104 abstract "resistance. This effect reversed near the reversal potential for K +, suggesting that it is mediated by a decrease in leak K + conductame. In thalamocortical cells, the TRH-induced depolarisation (+3.54-1.5 mV) was of sufficient amplitude to block the generation of rebound Ca 2+ spikes, while the even larger direct depolarisation (+134-5 mV) of PGN neurons transformed these cells from the burst to tonic, single spike mode of action potential generation. Furthermore, application of TRH prominently enhanced the afterdepolarisation following rebound Ca 2+ spikes (+454-23%; area trader curve), suggesting that this transmitter may also enhance Ca2+-activated non-specific (CAN) -currents. Conclusions: These data suggest a novel role for TRH in the brain as an excitatory, intrinsic regulator of thalamocortical network activity, and provide a potential mechanism for the previously demonstrated wake-promoting and antiepileptic effects of this peptide (Nishino et al., 1997)." @default.
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- W2056788104 date "2005-01-01" @default.
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- W2056788104 title "P.1.29 Subtype dependent changes of α1-adrenoceptormRNAs expression in the rat prefrontal cortex induced by the chronic mild stress procedure" @default.
- W2056788104 doi "https://doi.org/10.1016/s0924-977x(05)80134-8" @default.
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