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• W2152214053 abstract "The neuropeptide galanin is a modulator of cholinergic function and may play a role in A beta peptide-induced degeneration of cholinergic forebrain neurons. We have studied the effect of galanin and its galanin receptor subtype 2/3 agonist Gal2-11on toxicity induced by freshly-prepared beta-amyloid(25-35) in the cholinergic cell line SN56. Both nuclear fragmentation and caspase-3 expression were analysed. beta-amyloid(25-35)-exposure induced a significant increase in caspase-3 mRNA expression after 30, 60, 90 or 150 min of beta-amyloid(25-35) exposure. These effects were abolished in the presence of Gal2-11 (10 nM). Similarly, beta-amyloid(25-35)-induced nuclear fragmentation was prevented by the galanin agonist at all time points studied. These findings indicate that the galanin 2/3 agonist Gal2-11 protects SN56 cholinergic cells from beta-amyloid(25-35)-induced cell death and that this action is mediated by an early reduction of caspase-3 expression." @default.
• W2152214053 created "2016-06-24" @default.
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• W2152214053 date "2009-01-01" @default.
• W2152214053 modified "2023-10-09" @default.
• W2152214053 title "The galanin receptor 2/3 agonist Gal2-11 protects the SN56 cells against β-amyloid_25-35toxicity" @default.
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• W2152214053 doi "https://doi.org/10.1002/jnr.22278" @default.
• W2152214053 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/19885864" @default.
• W2152214053 hasPublicationYear "2009" @default.
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