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- W3037492032 endingPage "366" @default.
- W3037492032 startingPage "329" @default.
- W3037492032 abstract "Epilepsy is characterized by sudden and recurrent episodes of unprovoked excessive electrical activity in the brain, known as seizures. Seizures have a wide spectrum of phenotypes and are a common primary presenting symptom or chronic comorbidity in many neurological disorders. The leading mechanism underlying seizure generation has been historically described as an imbalance between excitation and inhibition. The majority of fast synaptic inhibition is mediated by GABAA receptors, chloride permeable pentameric ligand gated ion channels. The efficacy of GABAA receptor mediated inhibition is dependent upon the neuronal regulation of intracellular chloride concentration ([Cl−]i). SLC12A cation-chloride cotransporters, including the sodium-potassium-chloride cotransporter 1 (NKCC1) and the potassium-chloride cotransporter 2 (KCC2), regulate neuronal chloride homeostasis. The role of neuronal chloride transporters in the pathogenesis of seizures is discussed." @default.
- W3037492032 created "2020-07-02" @default.
- W3037492032 creator A5009879001 @default.
- W3037492032 creator A5021808184 @default.
- W3037492032 date "2020-01-01" @default.
- W3037492032 modified "2023-10-04" @default.
- W3037492032 title "The involvement of neuronal chloride transporter deficiencies in epilepsy" @default.
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