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• W988476802 abstract "While the evidence for the genetic basis of cancer is mounting [reviewed in 1-3], at the same time it is becoming apparent that growth factors and cytokines may play critical roles in determining the phenotypic expression of cancer [4-6]. Interleukin-6 (IL-6) decreases intercellular and substratum adhesiveness in lines of human ductal breast carcinoma cells [7-12]. Dyshesiveness is a cardinal pathological feature of breast carcinoma [13]. Ductal breast carcinoma cells are morphologically heterogeneous not only within the same tumor, but also in sublines originating from the same line of tumor cells. In the Ro subline of the ZR-75-1 cells EL-6 converts cuboidal/polygonal cells to stellate cells with long processes [7]. The morphologically altered ZR-75-1-Ro cells separate from each other and display enhanced directional motility although their proliferation is suppressed. No evidence was obtained of growth factor-inducible cell-cell separation with transforming growth factor-α (TGF-α), TGF-β1, epidermal growth factor (EGF), or insulin-like growth factor (IGF-1) [9]. Colonies of ZR-75-1-Ro cells formed in the presence of acidic fibro-blast growth factor (aFGF) or basic fibroblast growth factor (bFGF) (10 or 100 ng/ml) did however show some evidence of cell-cell separation." @default.
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• W988476802 date "1998-01-01" @default.
• W988476802 modified "2023-09-23" @default.
• W988476802 title "Cytokines in breast cancer cell dyshesion" @default.
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• W988476802 doi "https://doi.org/10.1007/978-3-0348-8946-9_2" @default.
• W988476802 hasPublicationYear "1998" @default.
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