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- W2054598045 abstract "Platelet activation and aggregation are considered to be central to arterial thrombus formation. Antiplatelet therapy is therefore important for both the treatment and prevention of cardiovascular disease. Aspirin, the most widely used antiplatelet agent, inhibits platelet cyclo-oxygenase and the conversion of arachidonic acid to the potent platelet agonist thromboxane A(2) but does not prevent platelet activation occurring via various signalling pathways that are independent of thromboxane A(2) release. Therefore a number of other compounds have been developed to complement aspirin's beneficial effect. These include the thienopyridines (clopidogrel and ticlopidine), dipyridamole, and the alpha(IIb)beta(3) (glycoprotein IIb/IIIa) receptor inhibitors." @default.
- W2054598045 created "2016-06-24" @default.
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- W2054598045 creator A5039450208 @default.
- W2054598045 date "2004-03-01" @default.
- W2054598045 modified "2023-09-27" @default.
- W2054598045 title "Antiplatelet therapy in cardiovascular disease" @default.
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- W2054598045 doi "https://doi.org/10.1136/pgmj.2003.007062" @default.
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