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• W2079771863 startingPage "518" @default.
• W2079771863 abstract "As we age, our arteries elongate and our brains sag. As a consequence of these processes, redundant arterial loops and bridging or intrinsic hindbrain veins may cause cross-compression of cranial nerve root entry zones in the cerebellopontine angle. This pulsatile compression can be seen to produce hyperactive dysfunction of the cranial nerve. Symptoms of trigeminal or glossopharyngeal neuralgia (somatic sensory), hemifacial spasm (somatic motor), tinnitus and vertigo (special sensory) and some cases of essential hypertension are caused by these vessels compressing cranial nerves V, IX--X, VII, VIII, and left X and medulla oblongata. Using microsurgical techniques, the symptoms may be relieved by vascular decompression, findings and results in 695 paients are briefly reviewed and correlated. A chronic primate model of essential hypertension is briefly described." @default.
• W2079771863 created "2016-06-24" @default.
• W2079771863 creator A5042157815 @default.
• W2079771863 date "1980-10-01" @default.
• W2079771863 modified "2023-10-06" @default.
• W2079771863 title "Neurovascular Compression in Cranial Nerve and Systemic Disease" @default.
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• W2079771863 doi "https://doi.org/10.1097/00000658-198010000-00010" @default.
• W2079771863 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/1346998" @default.
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