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- W2397848608 endingPage "345" @default.
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- W2397848608 abstract "Sepsis-associated encephalopathy (SAE), a complication of sepsis, is often complicated by acute and long-term brain dysfunction. SAE is associated with electroencephalogram pattern changes and abnormal neuroimaging findings. The major processes involved are neuroinflammation, circulatory dysfunction, and excitotoxicity. Neuroinflammation and microcirculatory alterations are diffuse, whereas excitotoxicity might occur in more specific structures involved in the response to stress and the control of vital functions. A dysfunction of the brainstem, amygdala, and hippocampus might account for the increased mortality, psychological disorders, and cognitive impairment. This review summarizes clinical and paraclinical features of SAE and describes its mechanisms at cellular and structural levels." @default.
- W2397848608 created "2016-06-24" @default.
- W2397848608 creator A5020719351 @default.
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- W2397848608 creator A5072996405 @default.
- W2397848608 creator A5074860086 @default.
- W2397848608 creator A5089010178 @default.
- W2397848608 date "2016-06-01" @default.
- W2397848608 modified "2023-10-16" @default.
- W2397848608 title "Neuroanatomy and Physiology of Brain Dysfunction in Sepsis" @default.
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