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- W2610391317 abstract "We attempt to demonstrate the regulatory role of miR-200c in glioma progression and its mechanisms behind. Here, we show that miR-200c expression was significantly reduced in the glioma tissues compared to paratumor tissues, especially in malignant glioma. Exogenous overexpression of miR-200c inhibited the proliferation and invasion of glioma cells. In addition, the in vivo mouse xenograft model showed that miR-200c inhibited glioma growth and liver metastasis, which is mainly regulated by targeting moesin (MSN). We demonstrated that the expression of MSN in glioma specimens were negatively correlated with miR-200c expression, and MSN overexpression rescued the phenotype about cell proliferation and invasion induced by miR-200c. Moreover, knockdown of MSN was able to mimic the effects induced by miR-200c in glioma cells. These results indicate that miR-200c plays an important role in the regulation of glioma through targeting MSN." @default.
- W2610391317 created "2017-05-12" @default.
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- W2610391317 date "2017-01-01" @default.
- W2610391317 modified "2023-09-30" @default.
- W2610391317 title "MiR-200c Inhibits the Tumor Progression of Glioma via Targeting Moesin" @default.
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- W2610391317 doi "https://doi.org/10.7150/thno.17886" @default.
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