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- W2955276558 endingPage "3224" @default.
- W2955276558 startingPage "3224" @default.
- W2955276558 abstract "Heart failure (HF) frequently coexists with atrial fibrillation (AF) and dysfunction of the sinoatrial node (SAN), the natural pacemaker. HF is associated with chronic adrenergic stimulation, neurohormonal activation, abnormal intracellular calcium handling, elevated cardiac filling pressure and atrial stretch, and fibrosis. Pulmonary veins (PVs), which are the points of onset of ectopic electrical activity, are the most crucial AF triggers. A crosstalk between the SAN and PVs determines PV arrhythmogenesis. HF has different effects on SAN and PV electrophysiological characteristics, which critically modulate the development of AF and sick sinus syndrome. This review provides updates to improve our current understanding of the effects of HF in the electrical activity of the SAN and PVs as well as therapeutic implications for AF." @default.
- W2955276558 created "2019-07-12" @default.
- W2955276558 creator A5002345532 @default.
- W2955276558 creator A5005288506 @default.
- W2955276558 creator A5020069509 @default.
- W2955276558 creator A5055156259 @default.
- W2955276558 creator A5063517419 @default.
- W2955276558 creator A5070376025 @default.
- W2955276558 date "2019-06-30" @default.
- W2955276558 modified "2023-10-05" @default.
- W2955276558 title "Heart Failure Differentially Modulates Natural (Sinoatrial Node) and Ectopic (Pulmonary Veins) Pacemakers: Mechanism and Therapeutic Implication for Atrial Fibrillation" @default.
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