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- W4310366944 abstract "The oral microbiome can initiate a range of bacterially driven diseases including caries and periodontal diseases. Among them, gingivitis and periodontitis are well-defined and common forms of periodontal diseases, being periodontitis is the progressive form of gingivitis and is associated with a range of systemic disease and driven by a hyperinflammatory phenotype. During periodontal inflammation, interleukin (IL)-1β activate endothelial cells and triggers adhesion of eosinophils increasing the inflammatory response. Several inflammasomes including Nod-like receptor (NLR) pyrin domain-containing protein 1 (NLRP1), NLRP2, NLRP3, NLR containing a caspase activating and recruitment domain (CARD) 4 (NLRC4) and Absent in melanoma 2 (AIM2) have been involved in the pathogenesis of periodontal diseases. Inflammasome modulation in diseases of the oral cavity is a potential novel therapeutic intervention for oral disease management." @default.
- W4310366944 created "2022-12-09" @default.
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- W4310366944 date "2023-01-01" @default.
- W4310366944 modified "2023-09-27" @default.
- W4310366944 title "Inflammasomes and their regulation in oral diseases" @default.
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- W4310366944 doi "https://doi.org/10.1016/b978-0-323-91802-2.00025-6" @default.
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