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Matches in Ubergraph for { <http://purl.obolibrary.org/obo/NCIT_C17916> ?p ?o ?g. }

• NCIT_C17916 IAO_0000115 "Breast cancer type 2 susceptibility protein (3418 aa, ~384 kDa) is encoded by the human BRCA2 gene. This protein plays a role in both cell cycle regulation and DNA repair." @default.
• NCIT_C17916 NCIT_NHC0 "C17916" @default.
• NCIT_C17916 NCIT_P100 "600185" @default.
• NCIT_C17916 NCIT_P106 "Amino Acid, Peptide, or Protein" @default.
• NCIT_C17916 NCIT_P107 "Breast Cancer Type 2 Susceptibility Protein" @default.
• NCIT_C17916 NCIT_P108 "Breast Cancer Type 2 Susceptibility Protein" @default.
• NCIT_C17916 NCIT_P207 "C2973986" @default.
• NCIT_C17916 NCIT_P322 "CTRP" @default.
• NCIT_C17916 NCIT_P366 "BRCA2_Protein" @default.
• NCIT_C17916 NCIT_P93 "P51587" @default.
• NCIT_C17916 NCIT_P98 "Encoded by human BRCA2 Gene, the highly charged 3,418-amino acid 384 kD putative tumor suppressor BRCA2 Protein is expressed (G1/S peak) in rapidly proliferating cells in breast, thymus, lung, ovary, and spleen. With no signal sequence, no membrane regions, and little similarity to other proteins, BRCA2 function is not known; a N-terminus similar to JUN activation domain is capable of activating transcription. BRCA2 may regulate cell proliferation and differentiation. BRCA2/BRCA1 may activate double-strand break repair or homologous recombination. BRCA2 may interact as a cofactor with RAD51 DNA-repair protein; the interaction with BRCA2 BRC3 or BRC4 regions blocks nucleoprotein filament formation. BRCA2 may also regulate the location and DNA-binding ability of RAD51. BRCA2 mutations are linked to elevated risk of breast cancer. (Swiss-Prot, OMIM)" @default.
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• NCIT_C17916 normalizedInformationContent "92.730304365937428" @default.
• NCIT_C17916 referenceCount "3" @default.
• NCIT_C17916 hasExactSynonym "BRCA1/BRCA2-Containing Complex, Subunit 2" @default.
• NCIT_C17916 hasExactSynonym "BRCA2 Protein" @default.
• NCIT_C17916 hasExactSynonym "BRCA2" @default.
• NCIT_C17916 hasExactSynonym "Breast Cancer 2 Tumor Suppressor" @default.

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