Matches in Ubergraph for { <https://frink.apps.renci.org/.well-known/genid/B4b669962db3089b1fd1acb98e78a4d2f> ?p ?o ?g. }
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- B4b669962db3089b1fd1acb98e78a4d2f hasDbXref 0000-0002-0736-9199 @default.
- B4b669962db3089b1fd1acb98e78a4d2f hasDbXref "PMID:19144691" @default.
- B4b669962db3089b1fd1acb98e78a4d2f hasDbXref "PMID:22449945" @default.
- B4b669962db3089b1fd1acb98e78a4d2f type Axiom @default.
- B4b669962db3089b1fd1acb98e78a4d2f annotatedProperty IAO_0000115 @default.
- B4b669962db3089b1fd1acb98e78a4d2f annotatedSource HP_0005576 @default.
- B4b669962db3089b1fd1acb98e78a4d2f annotatedTarget "A progressive detrimental connective tissue deposition (fibrosis) on the kidney parenchyma involving the tubules and interstitial tissue of the kidney. Tubulointerstitial injury in the kidney is complex, involving a number of independent and overlapping cellular and molecular pathways, with renal interstitial fibrosis and tubular atrophy (IF/TA) as the final common pathway. However, IF and TA are separable, as shown by the profound TA in renal artery stenosis, which characteristically has little or no fibrosis (or inflammation). For new annotations it is preferable to annotate to the specific HPO terms for Renal interstitial lfibrosis and/or Renal tubular atrophy." @default.