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• W100532170 abstract "In the human heart contractility and heart rate are regulated by receptor systems that act through accumulation of intracellular cAMP (GS-protein coupled), by receptor systems that act through inhibition of cAMP formation (Gi-protein coupled) and by receptor systems that act independently of cAMP formation possibly through the phospholipase C/diacylglycerol/inosito-1, 4,5-triphosphate (PLC/DAG/IP3)-pathway (Figure 1). Among all these receptors the s-adrenoceptor-Gs-protein-adenylate cyclase-cAMP system is the most powerful physiological mechanism to alter acutely contractility and heart rate. This chapter, deals with the properties of s-adrenoceptors in the human heart and their alterations in chronic heart failure." @default.
• W100532170 created "2016-06-24" @default.
• W100532170 creator A5063355903 @default.
• W100532170 date "1995-01-01" @default.
• W100532170 modified "2023-10-16" @default.
• W100532170 title "Heart Failure and the Cardiac Beta-Adrenoceptor" @default.
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• W100532170 doi "https://doi.org/10.1007/978-94-011-0023-6_14" @default.
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