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- W100945901 abstract "A large body of evidence suggests that there is an inflammatory component to Alzheimer’s disease (AD) pathogenesis. The reactive microglial cells of neuritic plaques are characterized by the presence of a variety of inflammatory markers, including HLA-DR, HLA-DP, HLA-DQ, β-2 integrins and Fc receptors (reviewed by Rogers et al., 1992a). Also evident within plaques are the Aß associated proteins (Selkoe, 1991), many of which are related to inflammatory processes. Some of the most studied are α 1-antichymotrypsin (ACT) (Abraham et al., 1988), complement proteins (McGeer and McGeer, 1992; McGeer and Rogers, 1992), serum amyloid P (SAP) (Coria et al., 1988) and heparin sulfate proteoglycans (HSPG) (Snow et al., 1988). Furthermore, altered levels of the cytokine interleukin 1 (IL-1) and IL-6 have been demonstrated in AD (reviewed in Wood et al., 1993)." @default.
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- W100945901 date "1994-01-01" @default.
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- W100945901 title "Inflammatory Pathology in Alzheimer’s Disease" @default.
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