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- W1031878387 abstract "Our understanding of the role of nitric oxide (NO) as an important paracrine regulator of vascular and microvascular responsiveness to vasoactive agonists is steadily growing. Since its discovery, numerous reports and reviews have been published describing the role of NO in virtually every organ system, including the kidney. Indeed, vascular resistance in the kidney may be more heavily influenced by endogenous NO production than in other organ systems. Studies have shown that renal vascular resistance is more sensitive to manipulation of NO synthesis than resistance in other vascular beds, such as the femoral, coronary and cerebral circulations (Sonntag et al. 1992; Sigmon et al. 1993). Several reviews have been written detailing the effect of NO on renal function and whole-kidney renal hemodynamics; however, less has been reported on the direct effects of NO on renal microvascular function. For a current assessment of the role of NO in the regulation of renal blood flow, the interested reader is referred to Chapter 19 by Majid and Navar as well as to several excellent reviews that have been published recently (Navar et al. 1996; Kone and Baylis 1997; Majid and Navar 1997; Mattson et al. 1997). This chapter will summarize what is currently known about the influence of endothelium-derived NO in regulating the renal microvasculature. Emphasis will be placed on detailing the direct effects of NO on renal microvascular responsiveness to vasoactive physiological agonists, tubuloglomerular feedback, and local renal microvascular control." @default.
- W1031878387 created "2016-06-24" @default.
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- W1031878387 date "2000-01-01" @default.
- W1031878387 modified "2023-09-28" @default.
- W1031878387 title "Modulation of Renal Microvascular Responsiveness by Nitric Oxide" @default.
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- W1031878387 doi "https://doi.org/10.1007/978-1-4612-1326-0_21" @default.
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