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• W1034709679 abstract "Unlike most metastatic tumors, carcinoids usually have a very slow rate of growth. Thus, the illness produced by carcinoid ncoplasms largely results from the pharmacologic effects of the tumors and patients with this syndrome may live for years with little morbidity directly related to the morphologic presence of the neoplasm. Thus, an understanding of the biochemical pathophysiology of the syndrome is important in developing a rationale for treating these patients. Although tumors, arising from organs, in the portal circulation produce carcinoid syndrome only after metastasizing to the liver, it is important that the increasing number of reports of surgical cures of carcinoid syndrome by resection of the extraportal primary tumors, such as bronchial adenomas and teratomas. The possibility that the pharmacologic effects of the tumor may occasionally signal the presence of a resectable malignancy makes early recognition and biochemical diagnosis of this syndrome imperative. Serotonin is the primary mediator of the endocrine aspects of the syndrome but not the sole mediator. Subsequent studies have shown that these tumors may vary in their metabolism of indoles and may elaborate entirely unrelated humoral agents, such as bradykinin, histamine, and adrenocorticotropic hormone (ACTH). Thus, within the broad classification of carcinoid tumors, there is a huge versatility in the production of biologically active substances." @default.
• W1034709679 created "2016-06-24" @default.
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• W1034709679 date "1967-01-01" @default.
• W1034709679 modified "2023-10-11" @default.
• W1034709679 title "Pharmacologic and Endocrine Aspects of Carcinoid Syndrome" @default.
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• W1034709679 doi "https://doi.org/10.1016/s1054-3589(08)60656-0" @default.
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