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- W1036003994 abstract "In some inflammatory diseases including rheumatoid arthritis (R.A.) and liver cirrhosis, it has been suggested that superoxide and peroxides can be generated in the tissues and accelerate the progression of the diseases. Since selenium is a component of the protective enzyme glutathione peroxidase (GSH-Px) we determined the serum levels of selenium in patient groups with such diseases. Selenium was determined by direct electrothermal atomic absorption after thermal stabilization of selenium compounds by the addition of nickel. The values found in healthy references (n=40) were 1.53 ± 0.25 μmol/l. The R.A. group (n=23) had slightly decreased selenium levels (1.19 ± 0.32 penal/1). Serum selenium in severe cases (n=11) of alcoholic cirrhosis was substantially decreased (0.64 ± 0.20 umol/l, whereas another patient group (n=1l) with the similar disease had 1.22 + 0.47 pmol/1. The values found in chronic active hepatis (n=15) and primary biliary cirrhosis (n-12) were 1.12 ± 0.27 pmol/1 and 1.37 ± 0.19 pmol/l, respectively. Selenium deficiency can lower the activity of OWN, e.g. in liver cells. Liver levels of selenium were low in cirrhotic patients. In the diseases investigated here, the levels of glutathione (OSH) and vitamin E, may also be decreased in the affected tissues, thus decreasing the protection toward peroxides. Following supplementation of R. A. patients with selenium the serum levels of selenium and OSH-Px were increased." @default.
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- W1036003994 date "1982-01-01" @default.
- W1036003994 modified "2023-10-11" @default.
- W1036003994 title "Selenium in Rheumatoid Arthritis and in Liver Cirrhosis" @default.
- W1036003994 doi "https://doi.org/10.1007/978-1-4612-5829-2_53" @default.
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