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- W1036732202 abstract "This chapter discusses the analysis of experimental glomerulonephritis. There exist several animal models that closely resemble various forms of human glomerulonephritis. The resemblance includes, in most instances, morphological, immunohistological, and pathophysiological similarities. The chapter also describes different methods for producing and studying three models that cover the spectrum of fixed and planted antigens and various mediator systems. Mediators of the glomerular injury induced by such immune deposits vary widely in different models. They include the complement (C) system acting either through its terminal, membranolytic pathway or via the inflammatory action of leukocytes; antibody-directed influx of leukocytes; and antibody alone. Host species, the nature and dose of antibody, and the distribution of antigen influence which mediators will operate in the various models studied to date. The redistribution of such glomerular epithelial cell antigens follows antibody binding; subsequent shedding of the resulting complexes is thought to account for the observed subepithelial electron densities." @default.
- W1036732202 created "2016-06-24" @default.
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- W1036732202 date "1988-01-01" @default.
- W1036732202 modified "2023-10-18" @default.
- W1036732202 title "[38] Experimental glomerulonephritis" @default.
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- W1036732202 doi "https://doi.org/10.1016/0076-6879(88)62096-9" @default.
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