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- W104307237 abstract "The striated muscle specific Lin 12, Islet 1, Mec 3 (LIM)-only protein MLP is a conserved positive regulator of myogenic differentiation associated with the actin-based cytoskeleton and the cell nucleus [1,2]. In the heart, MLP is expressed at high levels in atrial and ventricular myocytes during development and in the adult [1]. MLP consists of two LIM double-zinc fingers linked by a spacer of 58 residues. The LIM motif is a protein binding interface found in a diverse group of proteins [3]. MLP may promote myogenic differentiation by both acting as a molecular adapter to modulate protein assembly along the actin-based cytoskeleton, and by promoting muscle specific gene expression as a transcriptional co-factor. This hypothesis is supported by the demonstration that the second LIM motif of MLP can specifically target interacting proteins to the actin-based cytoskeleton [2], and the first LIM motif mediates the interaction of MLP with muscle-specific transcription factors [4]." @default.
- W104307237 created "2016-06-24" @default.
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- W104307237 date "1999-01-01" @default.
- W104307237 modified "2023-09-24" @default.
- W104307237 title "Mice Deficient in Muscle LIM Protein (MLP) Reveal a Pathway to Dilated Cardiomyopathy and Heart Failure" @default.
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- W104307237 doi "https://doi.org/10.1007/978-94-015-9321-2_4" @default.
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