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- W104392617 abstract "This chapter describes some studies on the genetic control of natural cytotoxic (NC) reactivity of different mouse strains and congenie partners against Meth 113 targets, which are a fibrosarcoma of BALB/c origin. Some mouse strains had normal high NC activity and low natural killer (NK) activity, including mice carrying the beige mutation. Although some variation of high–low reactivity within a strain has been observed with different targets for NC and NK, the screening of different mouse strains, especially congenie pairs, against a single prototype target has proven informative, both in NK and in NC studies. The results strongly suggest that at least three genes, probably all located in chromosome 17, distally from the D-end of the H2 region, determine the levels of NC reactivity in spleen against the Meth 113 target. None of the putative NC genes appear to affect NK activity. The genetic control of NC activity is different from that of NK activity. The genetic control of natural cell-mediated cytotoxicity is complex." @default.
- W104392617 created "2016-06-24" @default.
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- W104392617 date "1982-01-01" @default.
- W104392617 modified "2023-09-25" @default.
- W104392617 title "GENETIC CONTROL OF NC ACTIVITY IN THE MOUSE: THREE GENES LOCATED IN CHROMOSOME 17" @default.
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- W104392617 doi "https://doi.org/10.1016/b978-0-12-341360-4.50048-2" @default.
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