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- W1058066148 abstract "bystander cell apoptosis which was driven by paracrine signaling. Cleavage of cytokeratin 18 demonstrated apoptosis by 4 hours post infection, but the mechanism mediating this cleavage remains unknown as there was no activation of caspase-3. Inhibition of all caspase activity was able to prevent the early drop in TER, but not the later and more significant decline. CONCLUSIONS: C. parvumwill infect IPEC-J2 cells and complete asexual reproduction, but the infection is limited to a portion of the monolayer. Moreover, the apoptotic signaling in C. parvum infected IPEC-J2 cells is distinctly different than that of in vivo infection. These differences in signaling offer an opportunity to isolate the effect of subepithelial factors in C. parvum infection by comparing the IPEC-J2 cellular response to that of the piglet model." @default.
- W1058066148 created "2016-06-24" @default.
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- W1058066148 date "2013-05-01" @default.
- W1058066148 modified "2023-09-27" @default.
- W1058066148 title "Tu2014 Expression of EPEC MAP Is Significantly Different Than That of Other Type 3 Secreted Effectors In Vivo" @default.
- W1058066148 doi "https://doi.org/10.1016/s0016-5085(13)63370-3" @default.
- W1058066148 hasPublicationYear "2013" @default.
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