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• W10595887 abstract "1. Formiminoglutamic acid, a product of the catabolism of histidine, is excreted in abnormally large amounts in the urines of vitamin B12-deficient rats and of vitamin B12-deficient sheep; the excretion is reduced to negligible amounts after administration of vitamin B12. 2. After administration of certain methyl donors to vitamin B12-deficient rats or sheep urinary excretion of formiminoglutamic acid is temporarily decreased. 3. Irrespective of the pteroylglutamic acid status of the animals neither vitamin B12-deficient rats nor vitamin B12-deficient sheep have the ability to deal efficiently with histidine. 4. In sheep, urinary excretion of formiminoglutamic acid is increased after administration of aminopterin; treatment with pteroylglutamic acid restores the ability of the animal to deal with the catabolic products of histidine. 5. The possible functions of vitamin B12 and methionine in relieving a virtual deficiency of pteroylglutamic acid are discussed." @default.
• W10595887 created "2016-06-24" @default.
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• W10595887 date "1970-02-01" @default.
• W10595887 modified "2023-09-27" @default.
• W10595887 title "Factors affecting formiminoglutamic acid excretion in vitamin B12 deficiency" @default.
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• W10595887 doi "https://doi.org/10.1042/bj1160681" @default.
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