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- W106078148 abstract "The role of mitochondrial dysfunction has recently emerged as a potential contributing factor in epilepsy. Acute seizures result in free-radical production, oxidative damage to cellular targets, impaired mitochondrial redox status, mitochondrial dysfunction, and apoptosis. A limited number of recent studies have also demonstrated mitochondrial dysfunction in chronic epilepsies. Importantly, mitochondrial dysfunction is a potential cause of epileptic seizures associated with disorders resulting from mutations of mitochondrial DNA. In this article, we review the emerging role of mitochondrial dysfunction in epilepsy." @default.
- W106078148 created "2016-06-24" @default.
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- W106078148 date "2009-01-01" @default.
- W106078148 modified "2023-09-22" @default.
- W106078148 title "METABOLIC CHANGES | The Emerging Role of Mitochondrial Dysfunction and Oxidative Stress in Epilepsy" @default.
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- W106078148 doi "https://doi.org/10.1016/b978-012373961-2.00144-2" @default.
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