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- W112194451 abstract "This chapter provides an overview of antioxidant defense mechanisms and also describes the ultraviolet-induced skin inflammation. The skin provides a protective barrier over the body surface. As such, it is continuously exposed to injury by mechanical, infectious, and chemical means. Such insults cause an inflammatory response, which is usually acute and short term, but become chronic if physiological regulation of humoral and cellular response fails. The normal cellular response leads to migration of phagocytic leucocytes—macrophages, and polymorphonuclear leucocytes (PMNs)—and lymphocytes into the damaged site. During PMN phagocytosis in normal individuals, cells undergo a “respiratory burst” and consume increased amounts of oxygen. In the presence of catalytic iron, the highly reactive ͦOH radical is produced, which peroxidizes polyunsaturated fatty acids (PUFAs) in cell membranes. This is a particularly significant process in the skin that contains a very high concentration of cell-membrane PUFAs. The chapter discusses the role of phagocyte-derived reactive oxygen site (ROS) and the major importance of lipid peroxidation in skin inflammatory reactions in detail. It also discusses xanthine oxidase and nitric oxide in detail." @default.
- W112194451 created "2016-06-24" @default.
- W112194451 creator A5025147185 @default.
- W112194451 creator A5064525715 @default.
- W112194451 creator A5088920454 @default.
- W112194451 date "1995-01-01" @default.
- W112194451 modified "2023-09-27" @default.
- W112194451 title "Reactive Oxygen Species in Skin Inflammation" @default.
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