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- W113011608 endingPage "55" @default.
- W113011608 startingPage "33" @default.
- W113011608 abstract "This chapter overviews studies that have relied on knockout mice to elucidate the role of several cytokines in the pathogenesis of experimental autoimmune encephalomyelitis, collagen II-induced arthritis, insulin-dependent diabetes mellitus, myasthenia gravis, and autoimmune thyroiditis. A predominantly disease-promoting role of some cytokines (tumor necrosis factor, interferon-γ, interleukins-1, -4, -6, -12, and -18), and a predominantly disease-limiting role of other cytokines (interferon-β and interleukin-10) have been demonstrated. Thus, cytokine knockout mice have considerably helped to clarify the pathogenesis of models of autoimmune disease. Most strikingly, the mechanisms and the cell populations whose impacts were brought to light by these studies belong almost exclusively to the innate immunity compartment. This strengthens the view that disturbed recruitment of innate immunity mechanisms is at least as important in the pathogenesis of the autoimmune disease process as the antigen-specific reaction towards self." @default.
- W113011608 created "2016-06-24" @default.
- W113011608 creator A5032232927 @default.
- W113011608 creator A5054279562 @default.
- W113011608 creator A5071793915 @default.
- W113011608 date "2003-01-01" @default.
- W113011608 modified "2023-09-23" @default.
- W113011608 title "The Use of Cytokine Knockouts in Animal Models of Autoimmune Disease" @default.
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