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- W113384361 endingPage "70" @default.
- W113384361 startingPage "45" @default.
- W113384361 abstract "The failure of chemotherapy to achieve complete and durable responses has been attributed in large part to the phenomenon of drug resistance. In the laboratory, tumor cells can be selected for resistance to a particular cytotoxin by repeated exposure to that drug. The selected cells often display crossresistance to a number of functionally and structurally distinct drugs. This drug-resistant phenotype is referred to as multidrug resistance (MDR). Currently, the best understood mechanism of multidrug resistance is that which is mediated by the mdr 1 gene product, P-glycoprotein (P-gp). P-gp is a plasma membrane protein that functions as a multidrug transporter capable of effluxing a variety of antineoplastic agents, predominantly those derived from natural products (see tables 3-1). Cells which express P-gp accumulate less cytotoxin and, hence, are resistant to these drugs [1, 2, 3, 4, 5, 6]." @default.
- W113384361 created "2016-06-24" @default.
- W113384361 creator A5016706552 @default.
- W113384361 creator A5019649110 @default.
- W113384361 creator A5036188351 @default.
- W113384361 date "1995-01-01" @default.
- W113384361 modified "2023-10-18" @default.
- W113384361 title "The reversal of multidrug resistance" @default.
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