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- W1136783453 abstract "This chapter discusses the role of central nervous system in the regulation of synthesis and release of corticotrophin (ACTH). The chapter gives evidence in favor of a central noradrenergic system that inhibits ACTH secretion via a-receptorial stimulation. In dogs, the injection into the third ventricle of catecholamine precursors, catecholamines and drugs that release or protect catecholamines from inactivation, inhibits stress-induced adrenocortical activation. In rats, drugs that decrease brain catecholamine levels provoke adrenocortical activation, also when injected directly into the third ventricle of systemically ineffective doses. Specific depletion of noradrenaline but not of dopamine is able to cause increase of plasma corticosterone. Experiments concerning long-term catecholamine depletion, suggest the presence of a small, defined, functional pool of noradrenaline responsible for the tonic inhibition of ACTH secretion. Although a corticotrophin-releasing factor (CRF) is not chemically identified and synthesized, the neurovascular hypothesis of ACTH secretory cell regulation seems to be well founded. A possible role of brain catecholamines in the regulation of ACTH secretion is considered by numerous investigators." @default.
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- W1136783453 date "1973-01-01" @default.
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- W1136783453 title "Role of Brain Noradrenaline in the Tonic Regulation of Hypothalamic Hypophyscal Adrenal Axis" @default.
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- W1136783453 doi "https://doi.org/10.1016/s0079-6123(08)64075-3" @default.
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