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- W114920276 abstract "During the course of phagocytosis, normal polymorphonuclear leukocytes undergo remarkable alterations in oxidative metabolism which are insensitive to cyanide or azide. These events, collectively referred to as the respiratory burst, are listed in Table 1. Although neutrophils are equipped with a variety of bactericidal weapons, including non-oxidative processes such as hydrolytic enzymes and cationic proteins, several lines of evidence suggest that the respiratory burst plays a major role in the killing of Table 1The Respiratory BurstI. Increased oxygen consumptionII. Increased glucose oxidation via the hexose monophosphate shuntIII. Generation of hydrogen peroxideIV. Generation of superoxide anionV. Generation of chemiluminescenceVI. Reduction of tetrazolium dyesmany bacteria. Bacteria are ingested well under anaerobic conditions but most are not killed efficiently in this situation (1). Further, cells obtained from patients with chronic granulomatous disease fail to elicit a normal respiratory burst and are unable to adequately kill many types of microorganisms (2). Patients with this disease are highly susceptible to severe pyogenic bacterial infections which are frequently life-threatening." @default.
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- W114920276 date "1983-01-01" @default.
- W114920276 modified "2023-09-23" @default.
- W114920276 title "Oxidative Metabolism of Leukocytes and Its Relationship to Bactericidal Activity" @default.
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- W114920276 doi "https://doi.org/10.1007/978-1-4684-4481-0_3" @default.
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