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• W115026875 abstract "The retinoblastoma tumor suppressor (RB) was the first identified tumor suppressor based on germline predisposition to the pediatric eye tumor. Since these early studies, it has become apparent that the functional inactivation of RB is a common event in nearly all human malignancy. A great deal of research has gone into understanding how the loss of RB promotes tumor etiology and progression. Since malignant tumors are characterized by aberrant cell division, much of this research has focused upon the ability of RB to regulate the cell cycle by repression of proliferation-related genes. However, it is progressively understood that RB is an important mediator of multiple functions. One area that is gaining progressive interest is the emerging role for RB in regulating diverse features of immune function. These findings suggest that RB is more than simply a regulator of cellular proliferation; it is at the crossroads of proliferation and the immune response. Here we review the data related to the functional roles of RB on the immune system, relevance to immune evasion, and potential significance to the response to immune-therapy." @default.
• W115026875 created "2016-06-24" @default.
• W115026875 creator A5029801038 @default.
• W115026875 creator A5034535391 @default.
• W115026875 creator A5085023512 @default.
• W115026875 date "2015-12-17" @default.
• W115026875 modified "2023-09-27" @default.
• W115026875 title "The RB tumor suppressor at the intersection of proliferation and immunity: relevance to disease immune evasion and immunotherapy" @default.
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• W115026875 doi "https://doi.org/10.1080/15384101.2015.1010922" @default.
• W115026875 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4825764" @default.
• W115026875 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/25714546" @default.
• W115026875 hasPublicationYear "2015" @default.
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