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- W116317434 abstract "Male rats treated with large amounts of 17-α-ethinyl estradiol develop a pro-found hypolipidemia that affects all of the major lipoprotein classes (Chao et al. 1979). Plasma cholesterol, triglycerides, and the apolipoproteins A-I and E axe lowered to less than 10% of normal. The augmented clearance of low density lipoproteins (LDL) is eliminated in functionally eviscerated animals, indicating that the liver is the site of the increased removal (Chao et all979). This is in line with the observation that isolated perfused rat livers from est- radiol-treated rats catabolize LDL at a much higher rate than controls. Membranes, prepared from livers of estradiol-treated rats, exhibit a 3- to 10-fold increase in saturable binding sites for human LDL (Kovanen et al. 1979). The estradiol-stimulated lipoprotein receptor of the rat recognizes lipoproteins that contain apolipoprotein B-100 (LDL, VLDL, and VLDL remnants) in vivo and in vitro (Windier et al. 1980). The receptor also recognizes synthetic lamellar complexes of lecithin and apolipoprotein E as well as canine high density lipoproteins containing apolipoprotein E (Windier et al. 1980). It is biochemically and immunologically identical with the LDL-receptor of other species (Roach and Noel 1985; Cooper et al. 1987; Jaeckle et al. 1989)." @default.
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- W116317434 date "1993-01-01" @default.
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- W116317434 title "Induction of the Hepatic Uptake of Lipoproteins by Estrogen" @default.
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- W116317434 doi "https://doi.org/10.1007/978-3-642-84855-1_11" @default.
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