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• W11761243 abstract "EBV is unique among the human herpesviruses in its selective infection of B lymphocytes and epithelial cells. The tropism of EBV for B lymphocytes was shown by Jondal and Klein in 1973 (1), the same year that three laboratories independently described the existence of a B lymphocyte receptor for the C3d fragment of C3, the third complement component. This structure is now termed CR2. Studies carried out in the 1970’s by Klein, Jondal, Yefenof, Oldstone and collaborators showed a close relationship between the EBV and C3d receptors as they exhibited the same distribution on multiple cell types, capped together and independently of other cell surface antigens, reciprocally blocked binding in conjunction with anti-EBV or anti-C3, and were lost with EBV infection and reappeared with passage in certain cell lines (2,3). Subsequent analyses using other approaches further supported the association (reviewed in 4). Studies with newly developed monoclonal antibodies to CR2 strongly supported the identity of the two receptors." @default.
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• W11761243 date "1989-01-01" @default.
• W11761243 modified "2023-10-03" @default.
• W11761243 title "Cellular Receptors for Epstein-Barr Virus" @default.
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• W11761243 doi "https://doi.org/10.1007/978-1-4612-4508-7_59" @default.
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