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- W125587985 abstract "Examination of specific cholinergic and glutamatergic synapses reveals that nanomolar concentrations of nicotine potentiate both spontaneous and evoked transmission. Analysis of miniature excitatory synaptic currents (mepsc) before and after synaptic application of nicotine indicates that presynaptic changes underlie the synaptic facilitation, since nicotine increases mepsc frequency by more than 10 fold without altering the amplitude of the unit mode of mepsc histograms. Image analysis of changes in presynaptic [Ca]int, as well as pharmacological and antisense-mediated nicotinic acetylcholine receptor (nAChR) subunit deletion experiments suggest that synaptic transmission is potentiated by direct activation of high affinity presynaptic nAChRs that include the α7 subunit." @default.
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- W125587985 date "1995-01-01" @default.
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- W125587985 title "Presynaptic Nicotine-Gated Channels Potentiate Transmission at ACh and Glutamate-Mediated Synapses" @default.
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- W125587985 doi "https://doi.org/10.1007/978-3-0348-7445-8_15" @default.
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