Matches in SemOpenAlex for { <https://semopenalex.org/work/W126919170> ?p ?o ?g. }
- W126919170 endingPage "20" @default.
- W126919170 startingPage "1" @default.
- W126919170 abstract "It had long been considered that noradrenaline released from autonomic sympathetic nerves and classical humoral factors, including renin-angiotensin, vasopressin and adrenaline, regulate vascular tonus by directly contracting vascular smooth muscle. In other words, peripheral vascular resistance is maintained mostly by vasocontraction produced by these factors, and vasorelaxation is passively induced when their influence diminishes. Within the past two decades, however, the existence of definite active vasorelaxation mechanisms has been uncovered, e.g., peptidergic [1] and nitric oxidergic [2] nerves, vasorelaxing substances including atrial natriuretic peptides (ANP) [3], adrenomedullin [4], and endothelium-derived relaxing factors (EDRF) [5]. Vascular endothelium consists of only one layer of endothelial cells on the inner surface of the blood vessel and plays an important role as a barrier against blood cells directly contacting vascular smooth muscle cells, hence preventing blood coagulation. In addition to this influence, vascular endothelial cells have been revealed to regulate vascular tonus by producing and releasing potent vasoactive substances, endothelium-derived contracting factors (EDCF), as well as EDRF. A number of vasoactive substances exert not only short-term contracting or relaxing actions but also long-term hypertrophic or hyperplastic actions on cardiovascular tissues. Thus, cardiovascular structure and function appear to be controlled by a large number of endogenous vasoactive substances in a complicated manner." @default.
- W126919170 created "2016-06-24" @default.
- W126919170 creator A5079572703 @default.
- W126919170 date "1999-01-01" @default.
- W126919170 modified "2023-09-26" @default.
- W126919170 title "History of Endothelin" @default.
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