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- W129425891 abstract "The study of neurodegenerative diseases has begun converging with the previously disparate fields of free radical pathophysiology, excitotoxin-mediated neuronal death and mitochondrial physiology. This convergence has been prompted by increasing evidence that excitatory amino acids and free radical toxicity contribute to neuronal apoptosis (1) and to the etiology of chronic neurodegenerative diseases (2–12). Moreover, a growing body of evidence implicates mitochondrial energetic and oxidative dysfunction, due to congenital genetic defects, and perhaps to radical-induced mutations and oxidative enzyme impairment, in several neurodegenerative disorders (reviewed by [5,6,13–17])." @default.
- W129425891 created "2016-06-24" @default.
- W129425891 creator A5083275081 @default.
- W129425891 date "1999-01-01" @default.
- W129425891 modified "2023-10-11" @default.
- W129425891 title "Free Radicals and Mitochondria Dysfunction in Excitotoxicity and Neurodegenerative Disease" @default.
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