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- W130153342 abstract "Numerous evidence has demonstrated the involvement in growth control of interferon (IFN) regulatory factor-1 (IRF-1), which shows tumor suppressor activity. IRF-1 is a well-studied member of the IRF transcription factors that reveals functional diversity in the regulation of cellular response by activating expression of a diverse set of target genes, depending on the cell type and on the specific stimuli. IRF-1 gene rearrangements may be a crucial point in the pathogenesis of some cancer types. Furthermore, different aspects of the tumor suppressor function of IRF-1 may be explained, at least in part, by the observations that IRF-1 is a regulator of cell cycle and apoptosis and that its inactivation accelerates cell transformation. Studies on gene knockout mice contributed greatly to the clarification of these multiple IRF-1 functions. We summarize our current knowledge of the antigrowth effect of IRF-1, focusing also on a more general involvement of IRF-1 in mediating negative regulation of cell growth induced by numerous cytokines and other biologic response modifiers." @default.
- W130153342 created "2016-06-24" @default.
- W130153342 creator A5000169021 @default.
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- W130153342 creator A5040913442 @default.
- W130153342 creator A5056591202 @default.
- W130153342 creator A5081435652 @default.
- W130153342 date "2002-01-01" @default.
- W130153342 modified "2023-10-11" @default.
- W130153342 title "Review: IRF-1 as a Negative Regulator of Cell Proliferation" @default.
- W130153342 cites W1512072225 @default.
- W130153342 cites W1519148488 @default.
- W130153342 cites W1560895679 @default.
- W130153342 cites W1758311809 @default.
- W130153342 cites W1784799435 @default.
- W130153342 cites W1794193241 @default.
- W130153342 cites W1805856933 @default.
- W130153342 cites W1942680226 @default.
- W130153342 cites W1962468650 @default.
- W130153342 cites W1963547997 @default.
- W130153342 cites W1964322465 @default.
- W130153342 cites W1968713927 @default.
- W130153342 cites W1972535275 @default.
- W130153342 cites W1973912514 @default.
- W130153342 cites W1975324295 @default.
- W130153342 cites W1978031188 @default.
- W130153342 cites W1989262738 @default.
- W130153342 cites W1990017150 @default.
- W130153342 cites W1991595805 @default.
- W130153342 cites W1996856448 @default.
- W130153342 cites W2008228521 @default.
- W130153342 cites W2014237818 @default.
- W130153342 cites W2017190892 @default.
- W130153342 cites W2023553374 @default.
- W130153342 cites W2024868532 @default.
- W130153342 cites W2028297274 @default.
- W130153342 cites W2028850807 @default.
- W130153342 cites W2030528311 @default.
- W130153342 cites W2032361307 @default.
- W130153342 cites W2033212544 @default.
- W130153342 cites W2033417996 @default.
- W130153342 cites W2035722336 @default.
- W130153342 cites W2037772620 @default.
- W130153342 cites W2038541114 @default.
- W130153342 cites W2043663222 @default.
- W130153342 cites W2045891404 @default.
- W130153342 cites W2049049669 @default.
- W130153342 cites W2051260092 @default.
- W130153342 cites W2051316659 @default.
- W130153342 cites W2054194135 @default.
- W130153342 cites W2055952888 @default.
- W130153342 cites W2056079442 @default.
- W130153342 cites W2056970059 @default.
- W130153342 cites W2063651211 @default.
- W130153342 cites W206497858 @default.
- W130153342 cites W2073166594 @default.
- W130153342 cites W2077454531 @default.
- W130153342 cites W2077845541 @default.
- W130153342 cites W2078237614 @default.
- W130153342 cites W2079360957 @default.
- W130153342 cites W2084521363 @default.
- W130153342 cites W2088080321 @default.
- W130153342 cites W2090162429 @default.
- W130153342 cites W2096038076 @default.
- W130153342 cites W2099340972 @default.
- W130153342 cites W2102204735 @default.
- W130153342 cites W2106315019 @default.
- W130153342 cites W2108399976 @default.
- W130153342 cites W2110972790 @default.
- W130153342 cites W2112534911 @default.
- W130153342 cites W2129350136 @default.
- W130153342 cites W2136098448 @default.
- W130153342 cites W2139154967 @default.
- W130153342 cites W2144093304 @default.
- W130153342 cites W2147242002 @default.
- W130153342 cites W2154393492 @default.
- W130153342 cites W2155005693 @default.
- W130153342 cites W2157033538 @default.
- W130153342 cites W2160688175 @default.
- W130153342 cites W2162741492 @default.
- W130153342 cites W2163853656 @default.
- W130153342 cites W2245941149 @default.
- W130153342 cites W2259346194 @default.
- W130153342 cites W2312341819 @default.
- W130153342 cites W2313156944 @default.
- W130153342 cites W2314507989 @default.
- W130153342 cites W2318207983 @default.
- W130153342 cites W2319924232 @default.
- W130153342 cites W2329908871 @default.
- W130153342 cites W2564590762 @default.
- W130153342 cites W4230255947 @default.
- W130153342 cites W4247448391 @default.
- W130153342 cites W4251306929 @default.
- W130153342 cites W4362205050 @default.
- W130153342 doi "https://doi.org/10.1089/107999002753452647" @default.
- W130153342 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/11846974" @default.
- W130153342 hasPublicationYear "2002" @default.