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- W132207462 abstract "For over 50 years aldosterone excess has been known to raise blood pressure in humans (Conn's syndrome), and over 60 years ago the combination of deoxycorticosterone and salt was shown to do so in experimental animals. In both instances renal sodium and water retention was inculpated as the initial effector mechanism, with the resultant increase in circulatory volume followed by an increased cardiac output, in turn normalized by elevation of blood pressure. Subsequently, steroids with predominantly glucocorticoid activity were also shown to raise blood pressure, an effect that was not blocked by spironolactone, thus presumably mediated via glucocorticoid rather than mineralocorticoid receptors, and which appears to be mediated by reduced nitric oxide availability.1 In experimental animals, administration of high levels of testosterone causes hypertension, by blockade of the adrenal enzyme (CYP11B1) needed for glucocorticoid production, causing elevation of ACTH and deoxycorticosterone levels." @default.
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- W132207462 date "2007-01-01" @default.
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- W132207462 title "Aldosterone and Other Steroids" @default.
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