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- W135062248 abstract "Malignant tumours frequently cause hypercalcemia through both renal and skeletal mechanisms (Bonjour et al. 1988). Tumor-associated bone resorption can be localized, generalized, or both and is characterized clinically by destructive osteolytic bone metastases. It has become clear that tumors may produce several factors which stimulate osteoclastic bone resorption, leading to hypercalcemia, even in the absence of metastases to bone (Stewart et al. 1980; Mundy and Martin 1982; Mundy 1985; Burtis et al. 1988). Other mechanisms also contribute to the maintenance of elevated calcium levels. Hypercalcemia itself has complex effects on renal function and, apart from the impairment of glomerular filtration, enhances renal tubular calcium reabsorption (Hosking et al. 1981; Percival et al. 1985; Bonjour et al. 1988)." @default.
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- W135062248 date "1989-01-01" @default.
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- W135062248 title "Use of Clodronate and Calcitonin in Hypercalcemia Due to Malignancy" @default.
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- W135062248 doi "https://doi.org/10.1007/978-3-642-83668-8_3" @default.
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