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• W136435580 abstract "The aim of this critical review was to establish morphologic or biochemical diagnostic markers or indications for the pathogenesis of SIDS in the CNS and to review current literature dealing with CNS changes, especially in the brain stem. The functional disturbances recently discussed as the cause of SIDS, i.e. primary CNS changes in the sense of retardation of neurons in the brain stem, are presented first. Signs of retardation were obtained by quantification of dendritic spines [1, 2] and determination of phenylethanolamine-N-methyltransferase (PNMT), the adrenalin-synthesizing enzyme [3] and disturbances of endorphin immunoreactivity [4, 5]. Dendritic spine density was higher and PNMT activity weaker in the SIDS cases than in the controls, whereas no definitive findings of an increase or decrease in endorphin system activity were found. Signs of secondary CNS changes were established by quantification of the glial cell population in the brain stem [6–8] and determination of Purkinje cell density [9]; some authors found evidence of a brain stem gliosis, but no further indications of chronic systemic hypoxia or chronic hypoperfusion of the CNS could be established in SIDS victims." @default.
• W136435580 created "2016-06-24" @default.
• W136435580 creator A5040003373 @default.
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• W136435580 date "1990-01-01" @default.
• W136435580 modified "2023-09-24" @default.
• W136435580 title "Recent Neuropathologic Research in Sudden Infant Death Syndrome" @default.
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• W136435580 doi "https://doi.org/10.1007/978-3-642-75186-8_4" @default.
• W136435580 hasPublicationYear "1990" @default.
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