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- W137353486 abstract "This chapter focuses on studies of hyperalgesia following cutaneous injury. Hyperalgesia is a common manifestation of inflammation. It may manifest as pain in the absence of external stimuli—spontaneous pain, as pain that results from low-intensity ordinarily innocuous stimuli—allodynia, and as increased pain to noxious stimuli—injurious or potentially injurious. Information regarding the stimulus is signaled to the central nervous system by highly specialized types of sensory fibers. Cutaneous nociceptive afferent fibers can be unmyelinated (C fiber) or myelinated (A fiber). Sensitization of nociceptors is one neurophysiological correlate of hyperalgesia and is characterized by a decrease in threshold, an increased response to suprathreshold stimuli and, occasionally, by spontaneous activity. The endogenous chemicals that may play a part in hyperalgesia can be broadly subclassified into neurogenic and non-neurogenic. A variety of substances are released from such non-neural tissues as mast cells and vasculature, following injury. Hyperalgesia to mechanical stimuli may indicate certain chronic pain syndromes such as causalgia and reflex sympathetic dystrophy, as well as postherpetic neuralgia and metabolic neuropathies." @default.
- W137353486 created "2016-06-24" @default.
- W137353486 creator A5026711216 @default.
- W137353486 creator A5086565001 @default.
- W137353486 creator A5089858947 @default.
- W137353486 date "1990-01-01" @default.
- W137353486 modified "2023-09-25" @default.
- W137353486 title "Hyperalgesia and sensitization of primary afferent fibers" @default.
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