Matches in SemOpenAlex for { <https://semopenalex.org/work/W137510179> ?p ?o ?g. }
Showing items 1 to 51 of
51
with 100 items per page.
- W137510179 endingPage "561" @default.
- W137510179 startingPage "556" @default.
- W137510179 abstract "Neurofibromatosis is an autosomal dominant disorder caused by a mutation of a tumor supressor gene on the long arm of chromosome 17. There are two types of neurofibromatosis, and development of neurofibroma is one of clinical diagnostic criteria for neurofibromatosis. The clinical signs of neurofibromatosis include as skin lesions, bone deformities, and tumors involving central nervous system. About 25% of neurofibromatosis involves oral neurofibroma. Radiographically, oral neurofibroma is well-defined unilocular radiolucency, which involves mandibular canal, mandibular foramen and mental foramen. When a lesion is small and approachable, complete resection, including 1cm of marginal connective tissue, is feasible. However, there are studies reporting that the recurrence rate after surgical resection is high and frequent recurrence may even increase the risk of malignant transformation. This case reports a patient with neurofibromatosis type I, accompanying oral neurofibroma, who shows a favorable result after surgical resection of the oral lesion." @default.
- W137510179 created "2016-06-24" @default.
- W137510179 creator A5000642212 @default.
- W137510179 creator A5007966510 @default.
- W137510179 creator A5050822429 @default.
- W137510179 creator A5075548073 @default.
- W137510179 creator A5075894873 @default.
- W137510179 date "2008-08-31" @default.
- W137510179 modified "2023-10-12" @default.
- W137510179 title "ORAL MANIFESTATIONS OF NEUROFIBROMATOSIS TYPE 1: CASE REPORT" @default.
- W137510179 hasPublicationYear "2008" @default.
- W137510179 type Work @default.
- W137510179 sameAs 137510179 @default.
- W137510179 citedByCount "0" @default.
- W137510179 crossrefType "journal-article" @default.
- W137510179 hasAuthorship W137510179A5000642212 @default.
- W137510179 hasAuthorship W137510179A5007966510 @default.
- W137510179 hasAuthorship W137510179A5050822429 @default.
- W137510179 hasAuthorship W137510179A5075548073 @default.
- W137510179 hasAuthorship W137510179A5075894873 @default.
- W137510179 hasConcept C141071460 @default.
- W137510179 hasConcept C142724271 @default.
- W137510179 hasConcept C16005928 @default.
- W137510179 hasConcept C2777831590 @default.
- W137510179 hasConcept C2778102573 @default.
- W137510179 hasConcept C2778679214 @default.
- W137510179 hasConcept C2778984943 @default.
- W137510179 hasConcept C2781156865 @default.
- W137510179 hasConcept C2909023644 @default.
- W137510179 hasConcept C71924100 @default.
- W137510179 hasConceptScore W137510179C141071460 @default.
- W137510179 hasConceptScore W137510179C142724271 @default.
- W137510179 hasConceptScore W137510179C16005928 @default.
- W137510179 hasConceptScore W137510179C2777831590 @default.
- W137510179 hasConceptScore W137510179C2778102573 @default.
- W137510179 hasConceptScore W137510179C2778679214 @default.
- W137510179 hasConceptScore W137510179C2778984943 @default.
- W137510179 hasConceptScore W137510179C2781156865 @default.
- W137510179 hasConceptScore W137510179C2909023644 @default.
- W137510179 hasConceptScore W137510179C71924100 @default.
- W137510179 hasIssue "3" @default.
- W137510179 hasLocation W1375101791 @default.
- W137510179 hasOpenAccess W137510179 @default.
- W137510179 hasPrimaryLocation W1375101791 @default.
- W137510179 hasVolume "35" @default.
- W137510179 isParatext "false" @default.
- W137510179 isRetracted "false" @default.
- W137510179 magId "137510179" @default.
- W137510179 workType "article" @default.