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- W1412181864 endingPage "189" @default.
- W1412181864 startingPage "171" @default.
- W1412181864 abstract "The postsynaptic inhibition of spinal neurons, which is observed as a temporary decrease of excitability, is accompanied by a type of membrane hyperpolarization, the inhibitory postsynaptic potential (IPSP). IPSP is increased in magnitude when the membrane potential is lowered (depolarized) and reversed when the membrane is hyperpolarized. The reversal potential lies between the resting level of the membrane potential and the equilibrium potential for potassium ions. Intracellular injections of a variety of anions and cations indicate that some convert hyperpolarizing IPSPs into depolarizations. The amplitude of intracellularly recorded excitatory postsynaptic potentials (EPSPs) is reduced in the absence of any alteration in the postsynaptic membrane potential, conductance, or excitability. Assuming glycine as an inhibitory transmitter, the suppression of spinal inhibitory transmission by tetanus toxin results from the reduction in the amount of transmitter released by presynaptic impulses." @default.
- W1412181864 created "2016-06-24" @default.
- W1412181864 creator A5034119545 @default.
- W1412181864 date "1969-01-01" @default.
- W1412181864 modified "2023-10-09" @default.
- W1412181864 title "The Pharmacology of Spinal Postsynaptic Inhibition" @default.
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- W1412181864 doi "https://doi.org/10.1016/s0079-6123(08)63237-9" @default.
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