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- W141725635 abstract "Apoptosis plays an important role in organ development and repair, however, it is becoming clear that it may also be involved in a number of different diseases. In recent years, there has been growing evidence to implicate apoptosis in kidney development [1, 2] and various renal disorders including immune-mediated renal disease, polycystic kidney disease and acute tubular necrosis. Many of the proteins involved in the apoptotic mechanism are present in the kidney and their expression and/or activity are altered under different conditions and are associated with changes in structure and function. For example, the Fas receptor, which when stimulated by Fas, induces apoptosis [3, 4] is expressed in a number of renal cells including mesangial cells [5] and tubular epithelial cells [6]. Furthermore, renal Fas expression increases in rats following induction of endotoxemia and this is accompanied by evidence of apoptosis along the nephron [5]. Other important mediators of apoptosis are a novel family of cysteine proteases, termed caspases [7], and there is recent evidence indicating that there is an altered expression of members of this family in the rat kidney following ischemia reperfusion injury [8]." @default.
- W141725635 created "2016-06-24" @default.
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- W141725635 date "1999-01-01" @default.
- W141725635 modified "2023-09-26" @default.
- W141725635 title "Apoptosis in renal disease" @default.
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- W141725635 doi "https://doi.org/10.1007/978-3-0348-8741-0_13" @default.
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