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- W142053782 abstract "This chapter discusses the mechanisms and roles of apoptosis in pathology. Necrosis differs from apoptosis in structure, mechanism, and sequelae. Structurally, necrotic cells show critically damaged organelles, ruptured plasma membranes, and dispersal of cytoplasmic elements into the extracellular space. The mechanisms are various, but do not depend upon continuing synthetic activity. There is no evidence that specific signaling pathways are involved. There is breakdown of membrane homeostasis and net flow of water into the necrotic cell, whose density falls. Intracellular calcium rises uncontrollably to equilibriate with the millomolar concentrations in the extracellular space. The process results in an acute inflammatory reaction, triggered by complement-activating factors emanating from mitochondria that have escaped from the damaged cell. Alternatively, leukotrienes and other arachidonate chemotaxins can be generated from partially- degraded cell membranes. The arrival of neutrophil polymorphs permits digestion and phagocytosis of the constituents of the necrotic cells, but brings with it the risk of further tissue damage." @default.
- W142053782 created "2016-06-24" @default.
- W142053782 creator A5019378167 @default.
- W142053782 creator A5065530590 @default.
- W142053782 date "1991-01-01" @default.
- W142053782 modified "2023-10-14" @default.
- W142053782 title "Apoptosis: Mechanisms and Roles in Pathology" @default.
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