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- W142918489 abstract "Publisher Summary Dimethylarsinic acid (DMA) is known to have promoting activity on rat urinary bladder carcinogenesis in F344 rats initiated with N-butyl-N-(4-hydroxybutyl) nitrosamine (BBN). Sodium L-ascorbate is also a strong promoter in this animal model. (LewisxF344) F1 rats were used to compare the promoting activity between DMA and sodium L-ascorbate and to find molecular alterations in the urinary bladder tumors. Male, 6-week-old rats were given 0.05% BBN in drinking water for 4 weeks, and then the rats were kept with no treatment for group 1, administered 0.01% DMA in drinking water (group 2) or 5% sodium L-ascorbate in the powdered diet (group 3). Group 4 rats were continuously given BBN alone. At weeks 36 and 44, the rats were sacrificed and the urinary bladders were fixed in 10% phosphate buffered formalin and embedded in paraffin. HE however, DMA revealed weaker promotion activity than that of sodium L-ascorbate, although doses were different. LOH existed in the urinary bladder tumors treated with DMA, whereas no LOH was detected in the urinary bladder tumors treated with sodium L-ascorbate." @default.
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- W142918489 date "1999-01-01" @default.
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- W142918489 title "Differences of Promoting Activity and Loss of Heterozygosity Between Dimethylarsinic Acid and Sodium L-ascorbate in F1 Rat Urinary Bladder Carcinogenesis" @default.
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- W142918489 doi "https://doi.org/10.1016/b978-008043648-7/50030-3" @default.
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