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- W143514950 abstract "Flow reserve and NO bioavailability are blunted in the surviving hypertrophied left ventricle (LV) early after myocardial infarction (MI). During exercise, exacerbation of hemodynamic abnormalities, neurohumoral activation and perturbed NO bioavailability could exhaust coronary flow reserve and thereby impair myocardial O2 supply. We tested this hypothesis in swine with a 2–3 week old myocardial infarction (MI) and determined whether a loss of NO bioavailability contributes to impaired tissue perfusion during exercise." @default.
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- W143514950 date "2003-01-01" @default.
- W143514950 modified "2023-09-23" @default.
- W143514950 title "Alterations in Hemodynamic and Neurohumoral Responses to Exercise in Swine with Left Ventricular Remodeling Early after Myocardial Infarction: Role of Blunted NO Bioavailability" @default.
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- W143514950 doi "https://doi.org/10.1007/978-1-4419-9262-8_14" @default.
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