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- W145748862 abstract "Injury to the cardiovascular system causes an elevated expression of endothelin-1 (ET-1) and activation of several important signaling pathways including the mitogen-activated kinase (MAPK) cascade. The activation of these pathways has been implicated in the pathogenesis of cardiovascular disease caused by hypoxia, infections, and ischemia /reperfusion injury, cardiomyopathy and restenosis after balloon angioplasty. Important downstream targets of the MAPK and ET-1 pathways are the cell cycle regulatory molecules (cyclins, cyclin-dependent kinases, and cyclin-dependent kinase inhibitors). Regulation of these molecules contributes to remodeling throughout the cardiovascular system. In addition, cell cycle molecules are important in the regulation of angiogenesis. These new data have led to the development of potential therapeutic modalities targeting these regulatory molecules in order to ameliorate various cardiovascular disease states." @default.
- W145748862 created "2016-06-24" @default.
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- W145748862 creator A5053593712 @default.
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- W145748862 date "2000-01-01" @default.
- W145748862 modified "2023-09-25" @default.
- W145748862 title "Cell cycle molecules and diseases of the cardiovascular system" @default.
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- W145748862 doi "https://doi.org/10.2741/petkova" @default.
- W145748862 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/10762598" @default.
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