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- W145799106 abstract "Abuse of cocaine can produce many adverse effects, including convulsions and lethality, especially in overdose situations. Previous efforts to develop pharmacotherapies for cocaine overdose have been limited in success, and there are, currently no effective treatments for this medical emergency. Much of the immediate toxicity associated with a cocaine overdose results from overstimulation of the central and peripheral nervous systems. Resultant symptoms such as tachycardia, hypertension, hyperthermia, seizures, respiratory depression, and cardiovascular collapse, if severe enough, can lead to death. All of the aforementioned symptoms of a serious cocaine overdose involve processes that are regulated by glutamatergic systems (1–4), which appear overactivated in an overdose situation. Therefore, even if cocaine does not appear to directly bind to glutamate receptors, antagonism of these receptors is a viable drug development strategy for cocaine overdose by preventing and reversing endstage events such as convulsions, respiratory distress, and cardiovascular collapse that immediately precede death (Fig. 1)." @default.
- W145799106 created "2016-06-24" @default.
- W145799106 creator A5029132146 @default.
- W145799106 creator A5087856092 @default.
- W145799106 date "2002-01-01" @default.
- W145799106 modified "2023-09-23" @default.
- W145799106 title "Effects of Novel NMDA/Glycine-Site Antagonists on the Blockade of Cocaine-Induced Behavioral Toxicity in Mice" @default.
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- W145799106 doi "https://doi.org/10.1007/978-1-59259-306-4_16" @default.
- W145799106 hasPublicationYear "2002" @default.
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