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- W14779035 abstract "The hallmark of chronic renal disease is a decrease in glomerular filtration rate (GFR). The progressive loss of nephrons affects most of the functions of the kidney (Table 1). As GFR decreases, solutes that are excreted by the kidney preferentially by filtration (urea, creatinine) accumulate in body fluids and concentrations in plasma increase (1). Indeed, the plasma concentrations of urea and creatinine provide a crude measurement of the decrease in GFR. As GFR falls to values < 25% of normal ((30 mL/min), other solutes that are filtered and either reabsorbed or secreted by the renal tubules may accumulate in body fluids (1,2). These solutes include phosphate, sulfate, uric acid, magnesium, and hydrogen, the latter resulting in the development of metabolic acidosis. Finally, other compounds are retained in body fluids when renal disease is far advanced:phenols, guanidines, organic acids, indoles, a number of metabolic products, and certain peptides. Some of these compounds may be toxic above specific concentrations and could contribute to the symptoms and signs of advanced chronic renal insufficiency (uremia)." @default.
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- W14779035 date "2000-01-01" @default.
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- W14779035 title "Trace Element and Mineral Nutrition in Renal Disease" @default.
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- W14779035 doi "https://doi.org/10.1007/978-1-59259-040-7_16" @default.
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