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- W147806050 endingPage "314" @default.
- W147806050 startingPage "291" @default.
- W147806050 abstract "The stimulatory effect of glucagon on the conversion of liver glycogen to blood glucose was the first documented property of glucagon, the “hyperglycemic glycogenolytic factor” discovered as a contaminant in some commercial preparations of insulin. Sutherland (1950) has reviewed the early evidence that indicated the hormonal nature of glucagon, distinct from insulin, and that established the liver as the target for the hormone’s action. By the analysis of serial blood and liver samples from anesthetized dogs, Cahill et al. (1957) were able to link directly the glucagon-induced hyperglycemia with hepatic glycogenolysis. The effects of glucagon to increase both glucose output and glycogen breakdown in various isolated liver preparations may be well known; as experimental sophistication has grown over the years, such effects have been demonstrated in rabbit liver slices (Sutherland 1950), isolated perfused rat liver (SOKAL et al. 1964), isolated hepatocytes (Garrison and Haynes 1973; Seglen 1973; Wagle and Ingebretsen 1973), and liver cells in primary culture (Gerschenson and Casanello 1968; Walker and Grindle 1977). Numerous reports have stated similar observations, usually with liver preparations from rats or rabbits, but occasionally also from mice (Assimaco-poulos-Jeannet et al. 1973; Müller et al. 1976)." @default.
- W147806050 created "2016-06-24" @default.
- W147806050 creator A5077673040 @default.
- W147806050 date "1983-01-01" @default.
- W147806050 modified "2023-09-27" @default.
- W147806050 title "Glucagon and Liver Glycogen Metabolism" @default.
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