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- W1479683764 abstract "Summary The cerebral metabolic equivalent of the stagnant hypoxia of shock was produced in fifty subjects of seven different species of laboratory animal (dogs, calves, pigs, sheep, goat, rabbits, and rhesus monkeys). A benign perfusion technic was used to deliver autologous venous blood with a pO 2 of 35 mm Hg, while maintaining normal systemic arterial pO 2 , pressure, volume, and the like. The pathophysiologic complex of the acute respiratory distress syndrome developed in each. Newborn animals had a particularly fulminant course, with a pulmonary picture indistinguishable from that of clinical hyaline membrane disease. These pulmonary complications have a centrineurogenic basis. We postulate the sequence to be as follows: impaired cerebral oxidative metabolism with derangement of hypothalamic function; autonomically mediated, increased pulmonary venular resistance; engorged and hypertensive capillaries; hemorrhage and edema; inactivation of surfactant and atelectasis; and hyaline membranes. This appears to be a universal, species-independent phenomenon." @default.
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- W1479683764 date "1973-07-01" @default.
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- W1479683764 title "The centrineurogenic etiology of the acute respiratory distress syndromes" @default.
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- W1479683764 doi "https://doi.org/10.1016/s0002-9610(73)80090-x" @default.
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